1. The question whether amputation for lower femoral osteosarcoma should be by disarticulation of the hip or through the upper femur to leave a stump is discussed.

2. Sixty-eight such patients are reviewed. Thirty were treated by disarticulation and thirty-eight by through-femur amputation.

3. There was a 16 per cent incidence of stump recurrence after through-femur amputation, but none after disarticulation. Comparison of survival was not conclusive.

4. Disarticulation of the hip is advised.

1. A relatively simple method of occipito-cervical fusion using autogenous bone chips without internal fixation is described.

2. In patients with atlanto-axial subluxation posterior fusion from the occiput to the axis rather than from the atlas to the axis is more reliable and is preferred. Inclusion of the occiput adds no more than a few degrees to the restriction of movement that follows C. 1-2 fusion.

3. The indications for occipito-cervical fusion are discussed, particularly in relation to C. 1-2 instability in rheumatoid arthritis.

1 . A series of twelve patients with solitary lung metastases from primary tumours of bone is reviewed. There were seven osteosarcomas, two chondrosarcomas, two fibrosarcomas and one malignant chondroblastoma. In each patient the lung desposit was resected.

2. Four patients have so far died from the disease. The average survival of the others since pulmonary resection is six years and seven months.

3. It is suggested that the results of pulmonary resection in carefully selected patients with solitary pulmonary metastases fully justify the procedure.

4. Careful investigation to ensure as far as possible that the metastasis is solitary, and a "waiting period" in case others should develop, are essential if unnecessary surgery is to be avoided. A waiting period of three months is recommended.

An inquiry was made of ninety-seven patients with recurrent dislocation of the patella and forty patients with recurrent dislocation of the shoulder to see how often they had a relative similarly affected, and also how often such dislocation is associated with, and perhaps caused by, familial joint laxity.

Ten of those with recurrent dislocation of the patella and two of those with recurrent dislocation of the shoulder were found to have a near relative with a similar dislocation. Familial joint laxity was found in two of the ten families with more than one member affected by recurrent patellar dislocation, and in both those with more than one member with recurrent dislocation of the shoulder. Familial joint laxity was also found in two out of twenty patients with recurrent dislocation of the patella who had no family history of similar dislocation; but in none out of twenty patients with recurrent dislocation of the shoulder and who had no family history of similar dislocation.

Familial joint laxity may be the only cause of recurrent dislocation of the shoulder occurring in more than one member of the family. But there are other, as yet undefined, causes of familial recurrent dislocation of the patella.

1. The indications for amputation of all the toes for severe toe deformity in rheumatoid arthritis, hallux valgus and pes cavus are discussed.

2. The results of forty-seven amputations in twenty-eight patients are analysed: 93 per cent were found to be very satisfied with the operation.

3. The technique of the operation and the subsequent management are described. The importance of retaining full-length shoes fitted with metatarsal insoles and toe blocks to preserve a good gait and balance is emphasised.

The family we record draws attention to an association between recurrent dislocation of the patella and joint laxity, which is not confined to the knee. This may pass unrecognised if specific inquiry is not made. In this and other families reported, the joint laxity is inherited, as though due to a dominant gene, but some only of those affected suffer recurrent dislocation of the patella. It is probable that there are other genetically determined causes of recurrent patellar dislocation. In three other families we have seen more than one subject of patellar dislocation, but none had lax ligaments, and two other families have been recorded with no mention of associated joint laxity.

1. An account is given of fifty stress fractures of the fibula which occurred in athletes.

2. The characteristic symptoms, signs and radiological appearances are described, with details of treatment and prognosis.

3. The mechanism of the injury has been suggested on clinical grounds and supported by experimental methods.