Objective

The aim of this study was to assess the results of combined arthroscopically assisted posterior cruciate ligament reconstruction and open reconstruction of the posterolateral corner in patients with chronic (3 months or more) symptomatic instability and pain.

Introduction: The place of Anterior Cruciate Ligament (ACL) reconstruction in skeletally immature patients is now well established, but in reported series the numbers are few and the follow-ups short. Also, the majority of children are aged between 14 and 16 years and the results are as good as in adults. This report includes a number of ACL reconstructions in children aged less than 12 years.

Materials & Methods: A group of twenty patients with an average age of 13.5 years were reviewed. 7 children were aged 12 or under at the time of operation. There were 17 boys and 3 girls. The follow-up ranged from 12 to 72 months (mean 37.8 months).

Reconstruction was done by a standard 4-strand hamstring technique using an endobutton proximally and a spiked washer and screw distally in the tibia.

The IKDC, Lysholm and Tegner scores were used to assess the knees pre and post-operatively. Stability was measured using the KT-1000 arthrometer.

Results: Common modes of injury were football, rugby, skiing and squash. The left side was involved in 12 patients, and the right side in 8 patients. Interval between injury and surgery ranged from 3 to 22 months with an average of 8 months. Meniscal repair was carried out in 7 out of 12 patients. The average Tegner score before injury was 7.7, before operation was 4 and at the last follow-up was 7.6. The average pre-operative Lysholm score was 54.6 compared to the post-operative score of 93. There was no incidence of angular deformity or a limb length discrepancy. There has been 1 re-rupture in a child aged 11 years 11 months at operation but no meniscal injuries. The outcome in the 6 other children aged 12 or less at the time of operation has been as good as the older children.

Conclusion: Reconstruction of the anterior cruciate ligament using a trans-physeal technique gives good results in pre-pubertal children and in adolescents.

Background: It has been reported that there is poor correlation between neurological injury and degree of bony retropulsion in thoracolumbar burst fractures¹. Wilcox et al² showed biomechanically that there was poor concordance between the extent of post impact spinal canal occlusion and the maximum amount of occlusion that occurred at the moment of impact. In the current study we examined the possibility that variation in the termination of the conus medullaris may offer protection from neurological injury in a proportion of these fractures.

Methods: A retrospective study was made of 39 patients (26M:13M, mean age 35.9 years, range 15 – 75 years) presenting with a single level thoracolumbar burst fracture (T12–L2) between 1998 and 2001. A whole spine MRI scan was performed on all patients and the level of the conus noted. Age, sex, injury severity score (ISS), neurological status (ASIA motor score) and the transverse spinal canal area (TSCA) of the vertebral levels either side of the fractured vertebra was measured. A predicted TSCA for the injured level was then calculated from the mean of the TSCA’s of the adjacent levels. The actual TSCA of the injured level was calculated and this enabled a percentage decrease of the TSCA to be worked out from the predicted value. Analysis was made of the presence or absence of neurological injury in relation to canal compromise and involvement of the conus.

Results: Eighteen patients with neurological compromise and 21 with intact neurology (the age and sex distribution in the two groups were similar). The mean ± SD ASIA motor score of the patients studied was 90.4 ± 23. Mean ISS was 20.2 in the neurologically injured and 10.5 in the intact (p=0.0005). Mean TSCA of the canal was 218mm² in the intact and 150mm² in the injured groups (p=0.006) and mean %TSCA was 70 and 49 respectively (p=0.007). The conus lay between T12 and L2 in all patients. When the conus lay cranial to the fracture (n=13), 38% were neurologically intact. When the conus lay at the level of the fracture (n=26), 62% were intact (NS). Neurological deficit did not occur in the absence of neurological compression on MRI.

Conclusion: Our study showed that the risk of neurological injury from a thoracolumbar burst fracture was not decreased when the conus lay outside the fracture zone. However, there was a statistically significant difference in percentage of canal compromise when the patients with neurological impairment were compared with those that were neurologically intact.

Introduction: The poor correlation between neurological injury and degree of retropulsion in thoracolumbar burst fractures has been identified, but not adequately explained. We have examined the possibility that variation in the termination of the conus medullaris may offer protection from neurological injury in a proportion of these fractures.

Methods: A retrospective study was made of 39 patients presenting with single level thoracolumbar burst fractures between June 1998 and April 2001. Admission MRI was performed on all patients. Age, sex, ISS, neurological status, mode of treatment and any neurological recovery were recorded. From the MRI scans the levels of the conus and the fracture were noted. Transverse Spine Area(TSA) was measured at the cranial, caudal and injured levels. A predicted TSA and % TSA for the injury level was calculated from the mean of the two other levels. Analysis was of severity of neurological injury in relation to canal compromise and involvement of the conus.

Results: 26 male and 13 female patients of mean age 35.9 (SD 17) years and mean ASIA motor score 90.4 (SD 23) were studied. Neither sex nor age distribution differed between 18 neurologically injured and 21 intact patients. Mean ISS was 20.2 in the neurologically injured and 10.5 in the intact (p=0.0005). Mean TSA of the canal was 218mm2 in the intact and 150mm2 in the injured groups (p=0.006) and mean %TSA was 70 and 49 respectively (p=0.007). The conus lay between T12 and L2 in all. When the conus lay cranial to the fracture (n=13), 38% were neurologically intact. When the conus lay at the level of the fracture (n=26), 62% were intact (NS). Neurological deficit did not occur in the absence of neurological compression on MRI.

Conclusions: Neurological injury is not less likely when the conus lie outside the fracture zone. Canal compromise is a highly significant factor in neurological injury.