Aims: To clarify whether joint loading after meniscus repair causes increased pressure on the femoral condyle and is responsible for early cartilage damage. Methods: In sixteen human cadaver knees a bucket handle tear was created at the posterior horn of the medial and lateral meniscus. The lesion was repaired using two biodegradable implants (either Stingerª, Arrow¨, Dartª or Meniscal Screwª) on each occasion. Loading was compared to intact menisci and menisci after suture repair using 2/0 Ethibond. The specimens were mounted into specially designed jigs, taking all degree of freedom of the knee joint into account, and þxed to a material testing machine (Bionix 858 MTS). Constant loading of 350 N was performed during knee motion of 0¡Ð90¡ of ßexion. The meniscofemoral pressure was measured using the Tekscansystem. All þxation techniques were tested þve times. Results: Increased joint loading at the posterior horn occurred with increased ßexion angle of 0¡, 30¡, 60¡ and 90¡ of knee ßexion in the medial and lateral compartment (p< 0.05). No signiþcant increase in joint loading was noticed after meniscus repair with biodegradable implants. Conclusions: Biodegradable implants do not cause higher meniscofemoral joint loading due to meniscus implants in the posterior horn and resulting cartilage damage at the femoral condyle is unlikely.

The loss of full muscle activation contributes to weakness of the quadriceps muscle in patients with deficiency of the anterior cruciate ligament (ACL). We examined whether a deficit of voluntary activation (VA) of the quadriceps muscle can be reversed by reconstruction of the ACL and assessed its influence on muscle strength and clinical parameters.

We evaluated 12 male subjects with an isolated tear of the ACL and 12 matched control subjects before operation and two years after reconstruction of the ACL. Assessment included measurements of isometric knee-extension torque at maximal voluntary contraction (MVC force), knee stability tests, the International Knee Ligament Standard Evaluation Form and the Tegner activity score. A sensitive method of twitch interpolation was used to quantify the VA and to calculate true muscle force.

Before operation we found a deficit of VA on both the injured (mean ±SEM 74.9 ± 3.5%) and the uninjured side (74.6 ± 3.0%) in comparison with the control group (91 ± 0.9%). Two years after reconstruction of the ACL the VA improved significantly on both sides but remained less than that of the controls. Correlation analysis revealed an improvement of the VA in patients who returned to a higher level of activity. The deficit of true muscle force, however, persisted regardless of the clinical outcome and ligament stability.

We assessed proprioception using threshold levels for the perception of knee movement at slow angular velocities (0.1°/s to 0.85°/s) in 20 patients with unilateral tears of the anterior cruciate ligament (ACL) and 15 age-related control subjects. Failure to detect movement was also analysed.

The threshold levels of detection did not differ between the damaged and undamaged knees in the patients or between the patients and the control group. Failure to appreciate movement, however, was significantly greater in knees with ACL loss compared with the undamaged knees of patients and the control group.

Our findings show a proprioceptive deficit in the absence of the ACL. Measurements of threshold levels of detection of passive movement alone are not suitable for the evaluation of proprioceptive loss in ACL deficiency; assessment of failure to appreciate movement is essential.