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Orthopaedic Proceedings
Vol. 93-B, Issue SUPP_III | Pages 311 - 312
1 Jul 2011
Sharma A Seagrave M Fairbairn J Jeffcoate W Scammell B
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Background: The mechanisms underlying the increased prevalence of arterial calcification in diabetes are not understood. An association with distal neuropathy has been reported and a particularly high prevalence was found in patients with Charcot’s disease.

Aim: The aim of this study was to confirm this high prevalence and to determine whether it is specific to that disorder by comparing the results to patients with other types of foot disease.

Methods: A retrospective survey was conducted in three groups of patients with X-rays managed by a specialist service for the diabetic foot between 2002 and 2005. Group A (n=34) comprised patients with an acute Charcot foot, Group B (n=53) included patients with osteomyelitis and Group C (n=35) consisted of patients who had neither osteomyelitis nor Charcot’s disease. All X-rays were independently examined by three observers blinded to the underlying diagnosis, with films from each group being mixed.

Results: No differences existed (p> 0.05) in the mean age of the patients (60, 72 and 68 years, respectively), the proportion of men (68%, 64% and 51%) and the prevalence of nephropathy (41%, 30% and 14%). 100% patients in Group A, 94% in Group B and 80% of Group C had evidence of neuropathy. The overall prevalence of calcification in the three groups was 53%, 66% and 54% (p> 0.05). With all three groups combined, the only factor associated with calcification was disease duration (p=0.004). The prevalence of calcification was higher than the 40% previously reported in patients with neuropathy, but lower than that reported in patients with Charcot.

Conclusion: As there was no difference in the prevalence of calcification between the three groups, it is concluded that the increase is not specific to Charcot’s disease. It is possible that the increase in calcification in each group reflects the effect of local inflammation, possibly by activation of the RANKL/OPG signalling system.


Orthopaedic Proceedings
Vol. 90-B, Issue SUPP_II | Pages 359 - 360
1 Jul 2008
Aref Y Neumann L Fairbairn J
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Aim: To assess the outcome of arthroscopic subacromial decompression on rotator cuff muscles

Material: 44 patients with impingement syndrome and intact rotator cuffs were recruited 3 years ago. All were followed up 6 months after surgery and 17 were selected randomly to attended after 3 years.

Methods: All patients prior to surgery underwent:

Clinical examination.

Constant Scoring.

Cybex testing of the rotator cuff muscles.

MRI scans.

Arthroscopic examination followed by arthroscopic SAD.

Standard post-op rehab.

Tests were repeated at 6 months, and in 17 patients again 3 years after surgery.

Results: 1– 41patients (93.18%) improved subjectively at 6 months, and 12 (out of 17) were still satisfied after3 years. 3- Impingement signs disappeared in 41 patients at 6 months and in 13 out of 17 at three years. 4- Average increase in Constant score was 18 points at 6 months and 4 points at 3 years. Pain ADL Pos. ROM Power Total Pre-op mean 5.71 5.23 5.23 25.45 8.76 50.38 Post-op mean 11.67 8.04 8.14 38.05 20.9 86.8 3 years mean 10.91 8.02 7.83 42.78 16.6 86.14 5-Dynamometer (Cybex) Muscle testing: Average power in Watts Abduct Adduct Int. Rot. Ext. Rot. Int. Rot. Ext. Rotation at side at side at 90abduct at 90abduct Pre-op 40.01 61.3 38.2 23.12 32.56 27.78 6/12 postop 54.78 76.6 51.93 36.34 40.74 39.19 3 years p.o. 52.65 69.98 52.77 38.55 40.78 36.21

Conclusions

SAD still provides good pain relief

Muscle power increases post-operative but tends to plateau or slightly decrease after 3 years

Dynamic muscle power measurement is recommended for accurate assessment.

Other Cybex measurements (Torque) had little relation to clinical outcome.

Constant score does not accurately assess changes in muscle power.


Orthopaedic Proceedings
Vol. 85-B, Issue SUPP_I | Pages 72 - 72
1 Jan 2003
Aref Y Neumann L Fairbairn J
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Aim: To investigate the effect of Arthroscopic Subacromial Decompression and rehabilitation on rotator cuff muscle strength and function.

Material: 24 patients with primary impingement syndrome and intact rotator cuffs were recruited. 8 further patients were excluded due to pathologies found at arthroscopy.

Methods: All recruited patients prior to surgery underwent: 1- Through clinical examination. 2-Constant Scoring. 3- Detailed Cybex dynamometer testing of the rotator cuff muscles of both shoulders in six different positions with the same range and for power, speed and torque. 4- MRI scans. 5- Full arthroscopic examination followed by a routine arthroscopic SAD. 6-Standard post-op rehab programme.

All tests were repeated at the minimum of 6 (average 6.3) months after the operation.

Results:

CLINICAL

- 23 (95.83%) improved subjectively, and were back to their normal daily routines.

- The average improvement in VAS scoring was an average of 3.5 (2–7))

- Impingement signs disappeared in 23 patients (95.83%)

- Average increase in Constant score was 23 points, from 53.5 to 76.5.Average pain increased from 7.5 to 9, ADL from 5.5 to 6, hand position from 7 to 8, range of motion from 21.5 to 37 & power from 9 to 17. Dynamometer Muscle testing (All measurement of power was done in watts)

A-Abduction power increased from an average of 14.5 to 32.9, Adduction from 17 to 42

b- At maximal adduction internal rotators increased from 25.5 to 34.55, external rotators from 20.25 to 30.85 and

c- At 90° abduction the internal rotators increased from 19.55 to 31.3, and the external rotators from 16.6 to 21.95

Conclusions:

-ASD provides good pain relief

-Most patients return to work before the end of 6 months period; however there seems to be a tendency for further improvement after the 6 months period.

- Muscle power increases post-operative but with individual variation

-Dynamic Power assessment is preferable to the Constant Score method, as power is measured in Watts, and can test below 90° of elevation.

-Power measurement is the only parameter directly related to the clinical outcome of ASD.