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Orthopaedic Proceedings
Vol. 95-B, Issue SUPP_15 | Pages 145 - 145
1 Mar 2013
MacDessi S Chen D Seeto B Wernecke G
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AIM

Tibial component design has be been scrutinized in a number of studies in an attempt to improve tibial coverage in total knee arthroplasty. However, very few have controlled for both component rotation and resultant changes to posterolateral tibial tray overhang and posteromedial underhang. We hypothesize that asymmetrical tibial components can provide greater coverage than symmetrical trays without increasing overhang.

METHODS

The 6 most commonly used tibial trays on the Australian Joint Registry (2009) were superimposed on MRI slices of normal knees to assess tibial component overhang, underhang and percent coverage. Rotational alignment in this analysis was based upon the line joining the junciton of the medial and middle 1/3 of the patellar tendon and the PCL insertion.


Orthopaedic Proceedings
Vol. 95-B, Issue SUPP_15 | Pages 118 - 118
1 Mar 2013
MacDessi S Chen D Wernecke G Seeto B Chia S
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Introduction

Total knee arthroplasty is a painful operation. Peri-articular local anesthetic injections reduce post-operative pain and assist recovery. It is inconclusive whether intra-operative injections of peri-articular corticosteroids are of benefit. Clinical Question: In patients with osteoarthritis who are undergoing TKA, does the addition of high or low dose corticosteroid to peri-articular injections of local anesthetic and adrenaline improve post-operative pain and range of motion?

Methods

A prospective, randomized, double-blinded study was undertaken to assess the efficacy of adding peri-articular corticosteroids to intra-operative, peri-articular high volume local anaesthetic in post-operative pain management following TKA. 127 patients were randomised into three groups receiving local anaesthetic alone (control) or either low dose (40 mg) or high dose (80 mg) peri-articular corticosteroid plus local anaesthetic. Primary outcomes included ROM and visual analog pain scores (VAS). Pain was defined as the worst pain lasting for more than 20 minutes, measured at both rest (RVAS) and during activity (AVAS).


Orthopaedic Proceedings
Vol. 91-B, Issue SUPP_I | Pages 94 - 94
1 Mar 2009
Owers K Scougall P Dabirrahmani D Wernecke G Jhamb A Walsh W
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Negative ulnar variance, lunate shape and increased load transmission are associated with Kienbock’s disease. This may reflect trabecular alignment being more susceptible to shear forces along “fault planes” in Type 1 lunates, causing microfractures and avascular necrosis. The aim of this study was to assess the relationship between lunate bone structure, density and ulnar variance.

Standard 90/90 radiographs of 22 cadaveric wrists were taken for ulnar variance and lunate shape. The lunates were harvested and routine CT scans (1mm) were taken in 22/22 in the coronal, sagittal and transverse planes. DICOM files were analysed using Mimics (Materialise, Belgium) to measure Hounsfield units. MicroCT scans (SkyScan, Belgium) (40 μm) were taken in 10/22 in the coronal plane and measured for trabecular angle at the proximal and distal joint surfaces and the ‘tilting angle’ (between scaphoid and radius joint surfaces). Data was anlaysed using one-way ANOVA tests using SPSS for Windows.

Negative ulnar variance was noted in 7/22, neutral 10/22 and positive 5/22. Lunate shape according to Zapico was 0/22 Type 1, 18/22 Type 2 and 4/22 Type 3.

Lunate bone density was significantly lower in the ulnar positive specimens compared to ulnar negative and neutral (p< 0.001) (fig. 1).

The average trabecular angle measured 84.7° (+/− 4.5°) at the proximal and 90.3° (+/− 2.6°) at the distal joint surfaces and tilting angle was 115.7° (+/− 12.0°) (fig. 2). The 50% slice on the microCT correlated best with xray measurements of this angle.

This study quantifies the previous finding that load transmission through the lunate and hence lunate bone density is related to ulnar variance and that this is higher in ulnar negative wrists. MicroCT is a useful modality to assess trabecular structure and supports the ‘fault plane’ hypothesis of Kienbock’s Disease.