Abstract
Introduction
Many factors can influence post-operative kinematics after total knee arthroplasty (TKA). These factors include intraoperative surgical conditions such as ligament release or quantity of bone resection as well as differences in implant design. Release of the medial collateral ligament (MCL) is commonly performed to allow correction of varus knee. Precise biomechanical knowledge of the individual components of the MCL is critical for proper MCL release during TKA. The purpose of this study was to define the influences of the deep medial collateral ligament (dMCL) and the posterior oblique ligament (POL) on valgus and rotatory stability in TKA.
Materials and Methods
This study used six fresh-frozen cadaveric knees with intact cruciate ligaments. All TKA procedures were performed by the same surgeon using CR-TKA with a CT-free navigation system. Each knee was tested at 0°, 20°, 30°, 60°, and 90° of flexion. One sequential sectioning sequence was performed on each knee, beginning with an intact knee (S0), and thereafter femoral arthroplasty only (S1), tibial arthroplasty (S2), release of the dMCL (S3), and finally, release of the POL (S4). The same examiner applied all external load of 10 N-m valgus and a 5 N-m internal and external rotation torque at each flexion angle for the each cutting state. All data were analyzed statistically using one-way ANOVA and we investigated the correlation between the medial gap and the rotation angle. A significant difference was determined to be present for P < .05.
Results
There were no correlation between the medial gap and the rotation angle in S0. A moderate correlation was found in S1 at 0° and 20°, and a considerable correlation was found in S2 at 90°. There was a correlation at all angles in S4, and especially strong at 20°, 60°, 90°.
Conclusion
From this study, there were no correlation between medial knee instability and total rotation angles after performing TKA only by releasing dMCL, but by adding POL release, there were correlation in all angles. Therefore, medial knee instability caused by excessive release of the main medial knee structures may promote rotational instability.
