ARTHROFIBROSIS IN TKA

J. Boldt; U Munzinger

doi:10.1302/0301-620X.88BSUPP_I.0880111c

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ARTHROFIBROSIS IN TKA

7th Congress of the European Federation of National Associations of Orthopaedics and Traumatology, Lisbon - 4-7 June, 2005

J. Boldt
U Munzinger

ARTHROFIBROSIS IN TKA

Boldt J, Munzinger U. ARTHROFIBROSIS IN TKA. Orthop Procs. 2006;88-B(SUPP_I):111-111. doi:10.1302/0301-620X.88BSUPP_I.0880111c

Boldt, J., and U Munzinger. “ARTHROFIBROSIS IN TKA.” Orthopaedic Proceedings, vol. 88-B, no. SUPP_I, 2006, pp. 111-111., https://doi.org/10.1302/0301-620X.88BSUPP_I.0880111c

Boldt, J., & Munzinger, U. (2006). ARTHROFIBROSIS IN TKA. Orthopaedic Proceedings, 88-B(SUPP_I), 111-111. https://doi.org/10.1302/0301-620X.88BSUPP_I.0880111c

Boldt, J. and Munzinger, U. (2006) “ARTHROFIBROSIS IN TKA.” Orthopaedic Proceedings, 88-B(SUPP_I), pp. 111-111. Available at: https://doi.org/10.1302/0301-620X.88BSUPP_I.0880111c

Boldt, J., and U Munzinger. “ARTHROFIBROSIS IN TKA.” Orthopaedic Proceedings 88-B, no. SUPP_I (2006): 111-111. https://doi.org/10.1302/0301-620X.88BSUPP_I.0880111c

Boldt J, Munzinger U. ARTHROFIBROSIS IN TKA. Orthop Procs. 2006 Mar 1;88-B(SUPP_I):111-111. https://doi.org/10.1302/0301-620X.88BSUPP_I.0880111c

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Abstract

Aims: The purpose of this study was to determine whether internal mal-rotation of the femoral component is associated with arthrofibrosis in TKA. We hypothesized arthrofibrosis may be triggered by a combination of non-physiological kinematics (femoral component internal rotation) and a tight medial compartment.

Methods: From a consecutive cohort of 3058 mobile bearing TKA forty-four (1.4%) cases were diagnosed as having arthrofibrosis, of which thirty-eight (86%) cases could be recruited. Thirty-eight patients with a well functioning TKA served as matched controls. Evaluation included CT investigation to determine femoral component rotation with reference to the transepicon-dylar axis (TEA).

Results: Femoral components in the AF group were significantly (p< 0.00001) internally mal-rotated by a mean of 4.7 degrees ranging from ten degrees internal rotation (IR) to one degree external rotation (ER). Mean femoral rotational in the control group was parallel (0.3 degrees IR) to the TEA (six degrees IR to four degrees ER). Arthrofibrosis was not associated with age, gender, body-mass-index, or preoperative diagnosis.

Conclusions: There is a highly significant association between arthrofibrosis in TKA and internal mal-rotation of the femoral component. On the base of these results it was hypothesized that non-physiological kinematics in TKA with mal-aligned femoral components influence and/or trigger arthrofibrosis in TKA. In TKA with arthrofibrosis, we now consider femoral CT evaluation with the view to surgically rebalancing the flexion gap and realigning the femoral component, when internal mal-rotation is confirmed.

Theses abstracts were prepared by Professor Roger Lemaire. Correspondence should be addressed to EFORT Central Office, Freihofstrasse 22, CH-8700 Küsnacht, Switzerland.