Introduction: When subjected to strain or strain rates are higher than usual, the bone remodels to repair microdamage and to strengthen itself. During the initial resorption phase of remodeling, the bone is transitorily weakened and microdamage can accumulate leading to stress fracture.

Methods: To determine whether short –term suppression of bone turnover using bisphosphonates can prevent the initial loss of bone during the remodeling response to high bone strain and strain rates and potentially prevent stress fractures, we conducted a randomized, double blind, placebo-controlled trial of 324 new infantry recruits known to be at high risk for stress fracture. Recruits were given a loading dose of 30 mg of residronate or placebo daily for 10 doses during the first two weeks of basic training and then a once a week maintenance dose for following 12 weeks. Recruits were monitored by biweekly orthopedic examinations during 15 weeks of basic training for stress fractures. Bone scans for suspected tibial and femoral stress fractures and radiographs for suspected metatarsal stress fractures were used to verify stress fracture occurrence.

Results: By the intension to treat analysis and per protocol analysis, there was no statistically significant difference in the tibial, femoral, metatarsal, or total stress fracture incidence between the treatment group and the placebo.

Discussion: We conclude that prophylactic treatment with residronate in a training population at high risk for stress fracture using a maintenance dosage for the treatment of osteoporosis does not lower stress fracture risk.

Aims: To review the presentation, progression, treatment, and outcome of congenital posteromedial bow of tibia. Methods: Seventeen patients were studied using radiographs and medical records retrospectively. The time period was from 1989 to 2002. Data was collected with special reference to deformity correction and lengthening. Complications were analysed. Results: Eight of the patients were male and nine female; eleven of them had deformity correction and lengthening, whereas two are awaiting surgery, two underwent contralateral epiphysiodeses and one, periosteal stripping. Twelve were treated with the Ilizarov device, seven with LRS. The range of discrepancy pre – operatively was from 3 to 8.8 cm. Mean length gained was 3.7 cm with residual discrepancy within 0.6 cm. Complications noted were minor grades of infection and 3 patients required further corrective surgery. The bone-healing index was 62 days per cm. There was some decrease in ankle movement noted, this was unchanged or improved post-operatively. Conclusion: Successful simultaneous deformity correction and lengthening for this condition is possible. The Ilizarov frame provided more benefits in reducing complications. This is the largest series of lengthening and correction of this deformity published and the residual deformity and initial length discrepancy is greater than previously stated.